5/25/2025
Last update
5/25/2025
Reviewed By
Overview
SCIENTIFIC SCORE
Possibly Effective
Based on 13 Researches
7.3
USERS' SCORE
Good
Based on 7 Reviews
8.1
Supplement Facts
Serving Size: 1 Tablet
Amount Per Serving
%DV
Vitamin D3 (Cholecalciferol)
25 mcg (1,000 IU)
130%
Vitamin K K-2 (MK-4) (Menaquinone)
100 mcg
80%
Calcium (as dicalcium phosphate)
32 mg
2%
Top Medical Research Studies
9
We examined the impact of calcitriol, the active form of vitamin D, on heart attack recovery using a mouse model of myocardial infarction (MI). In our study, we treated mice that had suffered a MI with calcitriol and observed promising results.
Our findings revealed that calcitriol helped reverse negative effects on heart function and reduced cardiac remodeling after a heart attack. It also targeted the inflammatory response typically associated with MI, improving the survival of heart muscle cells and promoting their regeneration.
We discovered that calcitriol works by enhancing the activity of the Vitamin D receptor (VDR). This process not only interferes with inflammatory signals but also leads to positive changes at the genetic level, further supporting heart health after an MI. Overall, our study provides strong evidence of calcitriol's cardioprotective properties, making it a potential treatment avenue for better outcomes following a heart attack.
Our findings revealed that calcitriol helped reverse negative effects on heart function and reduced cardiac remodeling after a heart attack. It also targeted the inflammatory response typically associated with MI, improving the survival of heart muscle cells and promoting their regeneration.
We discovered that calcitriol works by enhancing the activity of the Vitamin D receptor (VDR). This process not only interferes with inflammatory signals but also leads to positive changes at the genetic level, further supporting heart health after an MI. Overall, our study provides strong evidence of calcitriol's cardioprotective properties, making it a potential treatment avenue for better outcomes following a heart attack.
Read More
9
We explored the impact of 1,25-dihydroxyvitamin D3 (VD3) on heart health, particularly after an acute myocardial infarction (AMI). To investigate this, we used male C57/BL6J mice and conducted a series of experiments, comparing those treated with VD3 to control groups.
Our findings revealed that VD3 treated mice showed significant improvement in heart function following AMI. This treatment not only enhanced cardiac function parameters but also reduced inflammation and the extent of myocardial damage. There was a notable decline in inflammatory markers and a decrease in cell apoptosis, indicated by a rise in protective proteins and a fall in those that lead to cell death.
In our in vitro studies with cardiomyocytes, we observed that VD3 boosted autophagy markers, enhancing the body’s ability to manage damaged cells. It seemed to activate key pathways involved in cell survival and repair, specifically the PI3K/AKT/mTOR pathway. However, when we inhibited this pathway with 3-methyladenine, the benefits of VD3 were reversed, indicating a direct link between autophagy promotion and the protective effects of VD3.
In summary, our research indicates that VD3 can be a valuable ally in the fight against heart damage and inflammation after a heart attack, primarily by supporting cellular repair processes.
Our findings revealed that VD3 treated mice showed significant improvement in heart function following AMI. This treatment not only enhanced cardiac function parameters but also reduced inflammation and the extent of myocardial damage. There was a notable decline in inflammatory markers and a decrease in cell apoptosis, indicated by a rise in protective proteins and a fall in those that lead to cell death.
In our in vitro studies with cardiomyocytes, we observed that VD3 boosted autophagy markers, enhancing the body’s ability to manage damaged cells. It seemed to activate key pathways involved in cell survival and repair, specifically the PI3K/AKT/mTOR pathway. However, when we inhibited this pathway with 3-methyladenine, the benefits of VD3 were reversed, indicating a direct link between autophagy promotion and the protective effects of VD3.
In summary, our research indicates that VD3 can be a valuable ally in the fight against heart damage and inflammation after a heart attack, primarily by supporting cellular repair processes.
Read More
8
We explored the impact of vitamin D levels on heart attack severity by examining 77 patients who experienced ST-elevation myocardial infarction (STEMI). A key focus was to understand how vitamin D deficiency might relate to the amount of thrombus, or blood clots, present in coronary arteries.
The findings revealed that a striking 79.22% of patients had vitamin D levels below what is considered adequate (less than 20 ng/mL). Notably, those with mild thrombus loads had higher vitamin D levels compared to those classified with severe thrombus loads, indicating a clear connection.
Furthermore, we discovered a negative correlation between vitamin D levels and the severity of thrombus. The lower the vitamin D levels, the higher the thrombus burden and the post-procedural TIMI frame count—essentially metrics that show how blood flows in the heart after treatment. This highlights that maintaining adequate vitamin D might be crucial for individuals at risk of severe heart complications.
The findings revealed that a striking 79.22% of patients had vitamin D levels below what is considered adequate (less than 20 ng/mL). Notably, those with mild thrombus loads had higher vitamin D levels compared to those classified with severe thrombus loads, indicating a clear connection.
Furthermore, we discovered a negative correlation between vitamin D levels and the severity of thrombus. The lower the vitamin D levels, the higher the thrombus burden and the post-procedural TIMI frame count—essentially metrics that show how blood flows in the heart after treatment. This highlights that maintaining adequate vitamin D might be crucial for individuals at risk of severe heart complications.
Read More
Most Useful Reviews
9
No TIA occurrences
I experienced frightening Trans Ischemic Attacks before using this product; now I haven’t had any since starting it. It's made a significant impact on my health.
Read More
7.5
Helpful for heart
5 people found this helpful
This is an outstanding supplement! It proves to be especially beneficial for individuals dealing with heart issues. I genuinely value its effects and will continue to share my positive experiences.
Read More
7.5
Relieves heart pain
1 people found this helpful
I bought this for my mother, and she reported less heart pain and more stable blood pressure since using it. It has made a noticeable difference in her well-being.
Read More
Medical Researches
SCIENTIFIC SCORE
Possibly Effective
Based on 13 Researches
7.3
- All Researches
9.5
We conducted a detailed study to uncover how vitamin D3, combined with exercise, affects recovery from heart attacks. Our research involved fifty-six male rats, some of which experienced a simulated heart attack, while others served as a control group.
The rats were then divided into several groups, receiving different treatments over eight weeks. We specifically looked at how vitamin D3 and aerobic-resistance training together impacted cardiac health, focusing on the important TGF-β1/Smad signaling pathway known to contribute to heart damage.
Our findings were quite revealing. We noticed that the combinations of vitamin D3 and exercise training significantly improved heart function. Specifically, those receiving both treatments showed higher heart ejection fractions and lower levels of TGF-β1 and collagen proteins, indicating less cardiac fibrosis. In contrast, the groups that only received one treatment did not show the same level of improvement.
This suggests that while vitamin D3 on its own was not studied in isolation, its combination with exercise led to better outcomes in heart attack recovery. Overall, these results indicate a promising role for vitamin D3 alongside exercise in supporting heart health after a heart attack.
The rats were then divided into several groups, receiving different treatments over eight weeks. We specifically looked at how vitamin D3 and aerobic-resistance training together impacted cardiac health, focusing on the important TGF-β1/Smad signaling pathway known to contribute to heart damage.
Our findings were quite revealing. We noticed that the combinations of vitamin D3 and exercise training significantly improved heart function. Specifically, those receiving both treatments showed higher heart ejection fractions and lower levels of TGF-β1 and collagen proteins, indicating less cardiac fibrosis. In contrast, the groups that only received one treatment did not show the same level of improvement.
This suggests that while vitamin D3 on its own was not studied in isolation, its combination with exercise led to better outcomes in heart attack recovery. Overall, these results indicate a promising role for vitamin D3 alongside exercise in supporting heart health after a heart attack.
Read More
9
We examined the impact of calcitriol, the active form of vitamin D, on heart attack recovery using a mouse model of myocardial infarction (MI). In our study, we treated mice that had suffered a MI with calcitriol and observed promising results.
Our findings revealed that calcitriol helped reverse negative effects on heart function and reduced cardiac remodeling after a heart attack. It also targeted the inflammatory response typically associated with MI, improving the survival of heart muscle cells and promoting their regeneration.
We discovered that calcitriol works by enhancing the activity of the Vitamin D receptor (VDR). This process not only interferes with inflammatory signals but also leads to positive changes at the genetic level, further supporting heart health after an MI. Overall, our study provides strong evidence of calcitriol's cardioprotective properties, making it a potential treatment avenue for better outcomes following a heart attack.
Our findings revealed that calcitriol helped reverse negative effects on heart function and reduced cardiac remodeling after a heart attack. It also targeted the inflammatory response typically associated with MI, improving the survival of heart muscle cells and promoting their regeneration.
We discovered that calcitriol works by enhancing the activity of the Vitamin D receptor (VDR). This process not only interferes with inflammatory signals but also leads to positive changes at the genetic level, further supporting heart health after an MI. Overall, our study provides strong evidence of calcitriol's cardioprotective properties, making it a potential treatment avenue for better outcomes following a heart attack.
Read More
9
We explored the effects of vitamin D3 on heart injury caused by ischemia/reperfusion (I/R), a common scenario during heart attacks. Using a laboratory model that mimicked this condition, we discovered that I/R treatment significantly harmed heart cells, leading to cell death and increased oxidative stress.
We observed that I/R conditions prompted an increase in mitochondrial fission and mitophagy—mechanisms that can worsen heart injury. However, when we introduced vitamin D3, it appeared to counteract these detrimental effects. Specifically, vitamin D3 decreased cell death and reduced harmful mitochondrial changes, suggesting a protective role for this vitamin.
In live mice undergoing I/R, we confirmed that vitamin D3 treatment effectively reduced not only apoptosis (cell death) but also the adverse changes in mitochondrial function and structure. Overall, our findings indicate that vitamin D3 could be an important ally in safeguarding the heart during a heart attack by maintaining the integrity of mitochondrial function.
We observed that I/R conditions prompted an increase in mitochondrial fission and mitophagy—mechanisms that can worsen heart injury. However, when we introduced vitamin D3, it appeared to counteract these detrimental effects. Specifically, vitamin D3 decreased cell death and reduced harmful mitochondrial changes, suggesting a protective role for this vitamin.
In live mice undergoing I/R, we confirmed that vitamin D3 treatment effectively reduced not only apoptosis (cell death) but also the adverse changes in mitochondrial function and structure. Overall, our findings indicate that vitamin D3 could be an important ally in safeguarding the heart during a heart attack by maintaining the integrity of mitochondrial function.
Read More
9
We explored the impact of 1,25-dihydroxyvitamin D3 (VD3) on heart health, particularly after an acute myocardial infarction (AMI). To investigate this, we used male C57/BL6J mice and conducted a series of experiments, comparing those treated with VD3 to control groups.
Our findings revealed that VD3 treated mice showed significant improvement in heart function following AMI. This treatment not only enhanced cardiac function parameters but also reduced inflammation and the extent of myocardial damage. There was a notable decline in inflammatory markers and a decrease in cell apoptosis, indicated by a rise in protective proteins and a fall in those that lead to cell death.
In our in vitro studies with cardiomyocytes, we observed that VD3 boosted autophagy markers, enhancing the body’s ability to manage damaged cells. It seemed to activate key pathways involved in cell survival and repair, specifically the PI3K/AKT/mTOR pathway. However, when we inhibited this pathway with 3-methyladenine, the benefits of VD3 were reversed, indicating a direct link between autophagy promotion and the protective effects of VD3.
In summary, our research indicates that VD3 can be a valuable ally in the fight against heart damage and inflammation after a heart attack, primarily by supporting cellular repair processes.
Our findings revealed that VD3 treated mice showed significant improvement in heart function following AMI. This treatment not only enhanced cardiac function parameters but also reduced inflammation and the extent of myocardial damage. There was a notable decline in inflammatory markers and a decrease in cell apoptosis, indicated by a rise in protective proteins and a fall in those that lead to cell death.
In our in vitro studies with cardiomyocytes, we observed that VD3 boosted autophagy markers, enhancing the body’s ability to manage damaged cells. It seemed to activate key pathways involved in cell survival and repair, specifically the PI3K/AKT/mTOR pathway. However, when we inhibited this pathway with 3-methyladenine, the benefits of VD3 were reversed, indicating a direct link between autophagy promotion and the protective effects of VD3.
In summary, our research indicates that VD3 can be a valuable ally in the fight against heart damage and inflammation after a heart attack, primarily by supporting cellular repair processes.
Read More
8
We examined the effects of marine n-3 fatty acids, alongside vitamin D3, on cardiovascular disease events, particularly heart attacks. In the VITAL trial, which involved nearly 26,000 healthy older adults, participants were given either a supplement of n-3 fatty acids or a placebo to see if it would lower the risk of significant heart events.
The findings indicated a modest, though not significant, benefit of n-3 fatty acid supplementation regarding heart attacks. Specifically, the study revealed that while there was a reduction in non-fatal heart attacks, there was no clear impact on stroke outcomes. Interestingly, those with lower fish consumption at the start showed a stronger benefit from the n-3 fatty acids compared to their counterparts who consumed more fish.
However, it's important to note that the evaluation did not find any substantial isolated effect of vitamin D3 on heart attacks. Overall, our study suggests that while n-3 fatty acids might have some positive effects, particularly in certain groups, the same cannot be confidently stated for vitamin D3 alone in preventing heart attacks.
The findings indicated a modest, though not significant, benefit of n-3 fatty acid supplementation regarding heart attacks. Specifically, the study revealed that while there was a reduction in non-fatal heart attacks, there was no clear impact on stroke outcomes. Interestingly, those with lower fish consumption at the start showed a stronger benefit from the n-3 fatty acids compared to their counterparts who consumed more fish.
However, it's important to note that the evaluation did not find any substantial isolated effect of vitamin D3 on heart attacks. Overall, our study suggests that while n-3 fatty acids might have some positive effects, particularly in certain groups, the same cannot be confidently stated for vitamin D3 alone in preventing heart attacks.
Read More
User Reviews
USERS' SCORE
Good
Based on 7 Reviews
8.1
- All Reviews
- Positive Reviews
- Negative Reviews
9
No TIA occurrences
I experienced frightening Trans Ischemic Attacks before using this product; now I haven’t had any since starting it. It's made a significant impact on my health.
Read More
7.5
Helpful for heart
5 people found this helpful
This is an outstanding supplement! It proves to be especially beneficial for individuals dealing with heart issues. I genuinely value its effects and will continue to share my positive experiences.
Read More
7.5
Relieves heart pain
1 people found this helpful
I bought this for my mother, and she reported less heart pain and more stable blood pressure since using it. It has made a noticeable difference in her well-being.
Read More
9
Inhibits coronary disease
Vitamin K2 effectively maintains vascular and heart health by preventing calcium deposits in blood vessels, which can lead to coronary heart disease. Its role in blood coagulation ensures that deficiency doesn't result in abnormal clotting. Additionally, it supports a healthy nervous system and may increase insulin production. I appreciate its anti-inflammatory properties and the antioxidant protection it provides to my nerve cells.
Read More
7.5
Great for bones
High-quality ingredients in this vitamin D and K2 supplement support bone and heart health remarkably. I take one tablet daily, and it ensures my bones absorb the vitamin effectively. The price is appropriate, and I highly recommend it for anyone looking to enhance their bone and heart support. I’m already planning to repurchase.
Read More
Frequently Asked Questions
6
Supports diabetes management
After a month of taking Vitamin D3 and K2, I’ve noticed positive effects, particularly in managing my heart and diabetes. Living in a tropical area limits my sun exposure, but these supplements seem to enhance my health significantly. I might consider a higher dosage of D3 for optimal benefits.
7.5
Relieves heart pain
1 people found this helpful
I bought this for my mother, and she reported less heart pain and more stable blood pressure since using it. It has made a noticeable difference in her well-being.
9
No TIA occurrences
I experienced frightening Trans Ischemic Attacks before using this product; now I haven’t had any since starting it. It's made a significant impact on my health.
6
Regulates blood pressure
Combining D3 and K2 has proven excellent for my heart health and immunity. The high-quality ingredients support my overall wellness and help me regulate blood pressure effectively.
7.5
Helpful for heart
5 people found this helpful
This is an outstanding supplement! It proves to be especially beneficial for individuals dealing with heart issues. I genuinely value its effects and will continue to share my positive experiences.
4
We examined how vitamin D3 levels relate to heart attack recovery, specifically focusing on left ventricular ejection fraction in patients who recently experienced myocardial infarction (MI). In our study, 80 individuals who had undergone primary percutaneous coronary intervention for MI provided blood samples 24 hours later for measuring levels of calcidiol and calcitriol, which are forms of vitamin D.
Our findings revealed that a staggering 75% of these patients had low levels of 25-OH vitamin D3, with only 9% maintaining proper levels. Notably, those with a left ventricular ejection fraction of less than 40% displayed significantly lower concentrations of both calcidiol and calcitriol. This suggests that low vitamin D3 levels may influence heart function in the early stages following a heart attack.
However, despite these observations, it remains unclear whether vitamin D3 supplementation can specifically improve heart function post-MI, as most existing studies are either limited or based on laboratory settings. Our research opens up important conversations about the significance of vitamin D3 in cardiac health, but further clinical trials are necessary to determine any definitive benefits of treatment.
Our findings revealed that a staggering 75% of these patients had low levels of 25-OH vitamin D3, with only 9% maintaining proper levels. Notably, those with a left ventricular ejection fraction of less than 40% displayed significantly lower concentrations of both calcidiol and calcitriol. This suggests that low vitamin D3 levels may influence heart function in the early stages following a heart attack.
However, despite these observations, it remains unclear whether vitamin D3 supplementation can specifically improve heart function post-MI, as most existing studies are either limited or based on laboratory settings. Our research opens up important conversations about the significance of vitamin D3 in cardiac health, but further clinical trials are necessary to determine any definitive benefits of treatment.
8
We explored the impact of vitamin D levels on heart attack severity by examining 77 patients who experienced ST-elevation myocardial infarction (STEMI). A key focus was to understand how vitamin D deficiency might relate to the amount of thrombus, or blood clots, present in coronary arteries.
The findings revealed that a striking 79.22% of patients had vitamin D levels below what is considered adequate (less than 20 ng/mL). Notably, those with mild thrombus loads had higher vitamin D levels compared to those classified with severe thrombus loads, indicating a clear connection.
Furthermore, we discovered a negative correlation between vitamin D levels and the severity of thrombus. The lower the vitamin D levels, the higher the thrombus burden and the post-procedural TIMI frame count—essentially metrics that show how blood flows in the heart after treatment. This highlights that maintaining adequate vitamin D might be crucial for individuals at risk of severe heart complications.
The findings revealed that a striking 79.22% of patients had vitamin D levels below what is considered adequate (less than 20 ng/mL). Notably, those with mild thrombus loads had higher vitamin D levels compared to those classified with severe thrombus loads, indicating a clear connection.
Furthermore, we discovered a negative correlation between vitamin D levels and the severity of thrombus. The lower the vitamin D levels, the higher the thrombus burden and the post-procedural TIMI frame count—essentially metrics that show how blood flows in the heart after treatment. This highlights that maintaining adequate vitamin D might be crucial for individuals at risk of severe heart complications.
8
We examined the effects of marine n-3 fatty acids, alongside vitamin D3, on cardiovascular disease events, particularly heart attacks. In the VITAL trial, which involved nearly 26,000 healthy older adults, participants were given either a supplement of n-3 fatty acids or a placebo to see if it would lower the risk of significant heart events.
The findings indicated a modest, though not significant, benefit of n-3 fatty acid supplementation regarding heart attacks. Specifically, the study revealed that while there was a reduction in non-fatal heart attacks, there was no clear impact on stroke outcomes. Interestingly, those with lower fish consumption at the start showed a stronger benefit from the n-3 fatty acids compared to their counterparts who consumed more fish.
However, it's important to note that the evaluation did not find any substantial isolated effect of vitamin D3 on heart attacks. Overall, our study suggests that while n-3 fatty acids might have some positive effects, particularly in certain groups, the same cannot be confidently stated for vitamin D3 alone in preventing heart attacks.
The findings indicated a modest, though not significant, benefit of n-3 fatty acid supplementation regarding heart attacks. Specifically, the study revealed that while there was a reduction in non-fatal heart attacks, there was no clear impact on stroke outcomes. Interestingly, those with lower fish consumption at the start showed a stronger benefit from the n-3 fatty acids compared to their counterparts who consumed more fish.
However, it's important to note that the evaluation did not find any substantial isolated effect of vitamin D3 on heart attacks. Overall, our study suggests that while n-3 fatty acids might have some positive effects, particularly in certain groups, the same cannot be confidently stated for vitamin D3 alone in preventing heart attacks.
9
We explored the impact of 1,25-dihydroxyvitamin D3 (VD3) on heart health, particularly after an acute myocardial infarction (AMI). To investigate this, we used male C57/BL6J mice and conducted a series of experiments, comparing those treated with VD3 to control groups.
Our findings revealed that VD3 treated mice showed significant improvement in heart function following AMI. This treatment not only enhanced cardiac function parameters but also reduced inflammation and the extent of myocardial damage. There was a notable decline in inflammatory markers and a decrease in cell apoptosis, indicated by a rise in protective proteins and a fall in those that lead to cell death.
In our in vitro studies with cardiomyocytes, we observed that VD3 boosted autophagy markers, enhancing the body’s ability to manage damaged cells. It seemed to activate key pathways involved in cell survival and repair, specifically the PI3K/AKT/mTOR pathway. However, when we inhibited this pathway with 3-methyladenine, the benefits of VD3 were reversed, indicating a direct link between autophagy promotion and the protective effects of VD3.
In summary, our research indicates that VD3 can be a valuable ally in the fight against heart damage and inflammation after a heart attack, primarily by supporting cellular repair processes.
Our findings revealed that VD3 treated mice showed significant improvement in heart function following AMI. This treatment not only enhanced cardiac function parameters but also reduced inflammation and the extent of myocardial damage. There was a notable decline in inflammatory markers and a decrease in cell apoptosis, indicated by a rise in protective proteins and a fall in those that lead to cell death.
In our in vitro studies with cardiomyocytes, we observed that VD3 boosted autophagy markers, enhancing the body’s ability to manage damaged cells. It seemed to activate key pathways involved in cell survival and repair, specifically the PI3K/AKT/mTOR pathway. However, when we inhibited this pathway with 3-methyladenine, the benefits of VD3 were reversed, indicating a direct link between autophagy promotion and the protective effects of VD3.
In summary, our research indicates that VD3 can be a valuable ally in the fight against heart damage and inflammation after a heart attack, primarily by supporting cellular repair processes.
9
We examined the impact of calcitriol, the active form of vitamin D, on heart attack recovery using a mouse model of myocardial infarction (MI). In our study, we treated mice that had suffered a MI with calcitriol and observed promising results.
Our findings revealed that calcitriol helped reverse negative effects on heart function and reduced cardiac remodeling after a heart attack. It also targeted the inflammatory response typically associated with MI, improving the survival of heart muscle cells and promoting their regeneration.
We discovered that calcitriol works by enhancing the activity of the Vitamin D receptor (VDR). This process not only interferes with inflammatory signals but also leads to positive changes at the genetic level, further supporting heart health after an MI. Overall, our study provides strong evidence of calcitriol's cardioprotective properties, making it a potential treatment avenue for better outcomes following a heart attack.
Our findings revealed that calcitriol helped reverse negative effects on heart function and reduced cardiac remodeling after a heart attack. It also targeted the inflammatory response typically associated with MI, improving the survival of heart muscle cells and promoting their regeneration.
We discovered that calcitriol works by enhancing the activity of the Vitamin D receptor (VDR). This process not only interferes with inflammatory signals but also leads to positive changes at the genetic level, further supporting heart health after an MI. Overall, our study provides strong evidence of calcitriol's cardioprotective properties, making it a potential treatment avenue for better outcomes following a heart attack.
References
- Olędzki S, Siennicka A, Maciejewska-Markiewicz D, Stachowska E, Jakubiak N, et al. Calcitriol Concentration in the Early Phase of Myocardial Infarction and Its Relation to Left Ventricular Ejection Fraction. Metabolites. 2024;14. 10.3390/metabo14120686
- Ogata S, Manson JE, Kang JH, Buring JE, Lee IM, et al. Marine n-3 Fatty Acids and Prevention of Cardiovascular Disease: A Novel Analysis of the VITAL Trial Using Win Ratio and Hierarchical Composite Outcomes. Nutrients. 2023;15. 10.3390/nu15194235
- Thompson B, Waterhouse M, English DR, McLeod DS, Armstrong BK, et al. Vitamin D supplementation and major cardiovascular events: D-Health randomised controlled trial. BMJ. 2023;381:e075230. 10.1136/bmj-2023-075230
- Bassuk SS, Manson JE. Marine omega-3 fatty acid supplementation and prevention of cardiovascular disease: update on the randomized trial evidence. Cardiovasc Res. 2023;119:1297. 10.1093/cvr/cvac172
- Uguz B, Oztas S, Zengin I, Topal D, Tiryakioglu SK, et al. Relationship between vitamin D deficiency and thrombus load in patients with ST-elevation myocardial infarction. Eur Rev Med Pharmacol Sci. 2022;26:7015. 10.26355/eurrev_202210_29885
- Yang S, Wang C, Ruan C, Chen M, Cao R, et al. Novel Insights into the Cardioprotective Effects of Calcitriol in Myocardial Infarction. Cells. 2022;11. 10.3390/cells11101676
- Şen Ö, Şen SB, Topuz AN, Topuz M. Vitamin D level predicts angiographic no-reflow phenomenon after percutaneous coronary intervention in patients with ST segment elevation myocardial infarction. Biomark Med. 2021;15:1357. 10.2217/bmm-2020-0689
- Mehdipoor M, Damirchi A, Razavi Tousi SMT, Babaei P. Concurrent vitamin D supplementation and exercise training improve cardiac fibrosis via TGF-β/Smad signaling in myocardial infarction model of rats. J Physiol Biochem. 2021;77:75. 10.1007/s13105-020-00778-6
- Lee TL, Lee MH, Chen YC, Lee YC, Lai TC, et al. Vitamin D Attenuates Ischemia/Reperfusion-Induced Cardiac Injury by Reducing Mitochondrial Fission and Mitophagy. Front Pharmacol. 2020;11:604700. 10.3389/fphar.2020.604700
- Wei YX, Dong SM, Wang YY, Zhang P, Sun MY, et al. Autophagy participates in the protection role of 1,25-dihydroxyvitamin D3 in acute myocardial infarction via PI3K/AKT/mTOR pathway. Cell Biol Int. 2021;45:394. 10.1002/cbin.11495
- Akkuş O, Topuz M, Öz F, Harbalıoğlu H, Koca H, et al. Impact of 25(OH)D3 on spontaneous reperfusion and SYNTAX score in patients with ST-elevation myocardial infarction. Turk Kardiyol Dern Ars. 2018;46:268. 10.5543/tkda.2018.49393
- Le TYL, Ogawa M, Kizana E, Gunton JE, Chong JJH. Vitamin D Improves Cardiac Function After Myocardial Infarction Through Modulation of Resident Cardiac Progenitor Cells. Heart Lung Circ. 2018;27:967. 10.1016/j.hlc.2018.01.006
- Şen Ö, Topuz M, Acele A, Akkuş O, Baykan AO, et al. The influence of plasma 25-(OH) vitamin D levels in acute ST elevation myocardial infarction. Cardiol J. 2017;24:677. 10.5603/CJ.a2017.0066
